Unsupported Speculation

Entertainment without Tenure


The Origin and Destruction of Black Creeper

How it’s caused, frequency division making it worse, and victory.


Image by ChatGPT. Symbolizes the mysterious multiplication and spread of a neurological anomaly.


Ah! Back to Black Creeper again. Christ on a crucifix, pardon my language.

To those of you who don’t know what this is, you are lucky. It is a phenomenon that happens in human brains which stems from three main sources.

First, it is parts of all regions of the brain that I call the Send/Receive subregions, and they are to allow one specific region to communicate directly to its mirror counterpart in the opposite hemisphere. Thus, a left temporal for speech can communicate directly with the other temporal on the right which is also for speech.

Second, it also stems from the ability that all neurons in the brain to self-learn new nerve pathways to connect to not only new destinations within a particular region, but also connect outside of the origin region to destinations within other regions as well.

Related to number two is that when neurons begin encoding their routing to connect to other neurons, they can also encode new configurations which allow that area of the brain to gain new abilities than what is supposed to be there by evolution.

For example, this is like having a part of the brain which picks up cups to drink from, THEN develop alternative routing between all those same neurons to act as a detector of opportunity against your opponent in the game of chess.

If you think of neurons as being networks of cells fixed in their locations in the brain and connected by lots of nerves, it is possible for parts of the brain to find a new way to utilize those fixed neurons and nerve connections for a new purpose, simply by passing information between that collection of cells in a new way via different routings through the available nerve pathways. This is like the evil stepfather of the ‘Neuroplasticity’ of therapy, and instead of being used to ease troubling things, instead usurps that same plasticity of the brain to some mindless and malign purpose.

And at last, three: What can possibly be worse now? Frequency division of some part of the brain which has developed a new function, and then that part of the brain is duplicated multifold, so that you now have a long ‘tentacle’ of misery made up of seven or eight of the same single, solitary bastard, except now there are seven or eight of them. Wonderful.

Now this doesn’t have a chance to begin before there is a cut-off of communication that occurs between two same types of brain regions. Because the subregions I mentioned allow a region of the brain to exchange information with its counterpart on the opposite side, a cut-off can occur if the opposite region ‘doesn’t like’ or detects something awry with the region that communicated with it. When this cut-off occurs, then at that point, the send/receive subregions of both regions have nothing to share or retrieve, and now there is a chance that those neurons self-develop alternative pathways between themselves for a purpose other than communicating with the counterpart region.

The number of neurons in each region’s send/receive subregion is about 800, and the trouble about this same neuroplasticity that allows our brain to learn a new skill is that when the same phenomena occurs, but in a way that we are not benefitted at all, then collections of connected neurons only exist to find routings that gain it rewards in the form of increased neural activity where the collection of neurons are located.

In other words, the neurons will notice when a signal sent through them a new way results in a sharp increase of electrical stimulation where they are, and then that new routing will be reinforced chemically in the neuron’s memory as a routing to reattempt again. With enough reinforcements of their chemical memory, the pathway that results in heightened activity will be reused again and again.

What sorts of things increase activity for collections of neurons? They could be glorious things or horrible things. Neurons don’t actually care. You see, in the evolved way the brain is supposed to function, the brain adapts in order to avoid punishment outcomes in our life, as well as seek out the same decisions and behaviors which resulted in past successes. This is different however when neurons begin finding alternative routing between themselves, and since the ONLY reward is increased activity for themselves, then one of these collections of neurons could LEARN to evoke terrible things, which then gain it activity sustained over a long period of time.

How is it stopped? ‘Neuroplasticity’, which is code for getting your brain to de-emphasize routing patterns between neurons which creates malignant activity, as well as reestablishing communication between all the send/receive subregions of each region so that they go back to their true purpose.

The send/receive subregions are like the head of the snake. When their routings get new encodings, then it is only by curing the head that you stop something malign and errant from being duplicated multiple times when the brain utilizes frequency division in order to think about something with more efficiency.

It is some cosmic irony that something extra learned by the brain which is NOT there by evolution will get duplicated when this new learned skill formed in a place which allows itself to be frequency divided by design. Send/receive subregions frequency divide so that regions of the brain can communicate with their counterparts regardless of the frequency each of these different levels resides at. But, duplicated with them could be accidentally created things of spectacular horror, and as I wrote above, instead of one of the blighters, you’ll have seven or eight. If all send/receive subregions grow new abilities, instead of eight of the blighters, you’ll have 64.

Image by ChatGPT. Truely a thing of horror to behold.




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